The Thrombopoietin Receptor, MPL, Is Critical for the Development of JAK2V617F-Induced Cardiovascular Disease in a Murine Model of Myeloproliferative Neoplasm
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Media type:
E-Article
Title:
The Thrombopoietin Receptor, MPL, Is Critical for the Development of JAK2V617F-Induced Cardiovascular Disease in a Murine Model of Myeloproliferative Neoplasm