• Medientyp: E-Artikel
  • Titel: Twin-arginine-dependent translocation of folded proteins
  • Beteiligte: Fröbel, Julia; Rose, Patrick; Müller, Matthias
  • Erschienen: The Royal Society, 2012
  • Erschienen in: Philosophical Transactions of the Royal Society B: Biological Sciences, 367 (2012) 1592, Seite 1029-1046
  • Sprache: Englisch
  • DOI: 10.1098/rstb.2011.0202
  • ISSN: 0962-8436; 1471-2970
  • Entstehung:
  • Anmerkungen:
  • Beschreibung: Twin-arginine translocation (Tat) denotes a protein transport pathway in bacteria, archaea and plant chloroplasts, which is specific for precursor proteins harbouring a characteristic twin-arginine pair in their signal sequences. Many Tat substrates receive cofactors and fold prior to translocation. For a subset of them, proofreading chaperones coordinate maturation and membrane-targeting. Tat translocases comprise two kinds of membrane proteins, a hexahelical TatC-type protein and one or two members of the single-spanning TatA protein family, called TatA and TatB. TatC- and TatA-type proteins form homo- and hetero-oligomeric complexes. The subunits of TatABC translocases are predominantly recovered from two separate complexes, a TatBC complex that might contain some TatA, and a homomeric TatA complex. TatB and TatC coordinately recognize twin-arginine signal peptides and accommodate them in membrane-embedded binding pockets. Advanced binding of the signal sequence to the Tat translocase requires the proton-motive force (PMF) across the membranes and might involve a first recruitment of TatA. When targeted in this manner, folded twin-arginine precursors induce homo-oligomerization of TatB and TatA. Ultimately, this leads to the formation of a transmembrane protein conduit that possibly consists of a pore-like TatA structure. The translocation step again is dependent on the PMF.
  • Zugangsstatus: Freier Zugang